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Cascade of events resulting from amyloid-beta accumulation in the brain, including tau hyperphosphorylation, synaptic dysfunction, and neuronal death, alongside phytochemicals that may mitigate these pathological effects.

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![Figure 8: Cascade of events resulting from amyloid-beta accumulation in the brain, including tau hyperphosphorylation, synaptic dysfunction, and neuronal death, alongside phytochemicals that may mitigate these pathological effects.]()

> Source: Hamid Mostafavi Abdolmaleky et al. "Underlying Mechanisms of Brain Aging and Neurodegenerative Diseases as Potential." *Nutrients*, 2023. PMID: [37571393](https://pubmed.ncbi.nlm.nih.gov/37571393/)
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  <img src="" alt="Cascade of events resulting from amyloid-beta accumulation in the brain, including tau hyperphosphorylation, synaptic dysfunction, and neuronal death, alongside phytochemicals that may mitigate these pathological effects." />
  <figcaption>Figure 8. Cascade of events resulting from amyloid-beta accumulation in the brain, including tau hyperphosphorylation, synaptic dysfunction, and neuronal death, alongside phytochemicals that may mitigate these pathological effects.<br>  Source: Hamid Mostafavi Abdolmaleky et al. "Underlying Mechanisms of Brain Aging and Neurodegenerative Diseases as Potential." <em>Nutrients</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37571393/">37571393</a></figcaption>
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