Skip to main content
BrainCited

Açıklama

Neuroplasticity deficits in Alzheimer's disease and the role of PI3K-AKT signaling in tau hyperphosphorylation. The illustration shows how impaired signaling facilitates GSK3-beta-mediated pathology and how kaempferol/quercetin may intervene.

Cite This Figure

![Figure 10: Neuroplasticity deficits in Alzheimer's disease and the role of PI3K-AKT signaling in tau hyperphosphorylation. The illustration shows how impaired signaling facilitates GSK3-beta-mediated pathology and how kaempferol/quercetin may intervene.]()

> Source: Claire Alexander et al. "Polyherbal and Multimodal Treatments: Kaempferol- and Quercetin-Rich Herbs Allev." *Biology*, 2023. PMID: [37998052](https://pubmed.ncbi.nlm.nih.gov/37998052/)
<figure>
  <img src="" alt="Neuroplasticity deficits in Alzheimer's disease and the role of PI3K-AKT signaling in tau hyperphosphorylation. The illustration shows how impaired signaling facilitates GSK3-beta-mediated pathology and how kaempferol/quercetin may intervene." />
  <figcaption>Figure 10. Neuroplasticity deficits in Alzheimer's disease and the role of PI3K-AKT signaling in tau hyperphosphorylation. The illustration shows how impaired signaling facilitates GSK3-beta-mediated pathology and how kaempferol/quercetin may intervene.<br>  Source: Claire Alexander et al. "Polyherbal and Multimodal Treatments: Kaempferol- and Quercetin-Rich Herbs Allev." <em>Biology</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37998052/">37998052</a></figcaption>
</figure>