Descripción

Cell viability assays in neuronal cells exposed to amyloid-beta demonstrate dose-dependent protection by BDMC. Higher BDMC concentrations are associated with greater neuroprotection against amyloid toxicity.

Figure 4

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Source Paper

Bisdemethoxycurcumin inhibits oxidative stress and antagonizes Alzheimer's disease by up-regulating SIRT1.

Brain and behavior (2020)

PMID: 32441492

DOI: 10.1002/brb3.1655

Cite This Figure

![Figure 4: Cell viability assays in neuronal cells exposed to amyloid-beta demonstrate dose-dependent protection by BDMC. Higher BDMC concentrations are associated with greater neuroprotection against amyloid toxicity.]()

> Source: Yan Xu et al. "Bisdemethoxycurcumin inhibits oxidative stress and antagonizes Alzheimer's disea." *Brain and behavior*, 2020. PMID: [32441492](https://pubmed.ncbi.nlm.nih.gov/32441492/)

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  <img src="" alt="Cell viability assays in neuronal cells exposed to amyloid-beta demonstrate dose-dependent protection by BDMC. Higher BDMC concentrations are associated with greater neuroprotection against amyloid toxicity." />
  <figcaption>Figure 4. Cell viability assays in neuronal cells exposed to amyloid-beta demonstrate dose-dependent protection by BDMC. Higher BDMC concentrations are associated with greater neuroprotection against amyloid toxicity.<br>  Source: Yan Xu et al. "Bisdemethoxycurcumin inhibits oxidative stress and antagonizes Alzheimer's disea." <em>Brain and behavior</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32441492/">32441492</a></figcaption>
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