Description

Liver histopathology from bile duct-ligated mice showing fibrosis and cholestasis, confirming the development of chronic liver disease underlying the hepatic encephalopathy phenotype.

Figure 7

Micrograph

Source Paper

A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammonia overload, glial cell activation and neuroinflammation.

Scientific reports (2022)

PMID: 36266427

DOI: 10.1038/s41598-022-22423-6

Cite This Figure

![Figure 7: Liver histopathology from bile duct-ligated mice showing fibrosis and cholestasis, confirming the development of chronic liver disease underlying the hepatic encephalopathy phenotype.]()

> Source: Wouter Claeys et al. "A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammoni." *Scientific reports*, 2022. PMID: [36266427](https://pubmed.ncbi.nlm.nih.gov/36266427/)

<figure>
  <img src="" alt="Liver histopathology from bile duct-ligated mice showing fibrosis and cholestasis, confirming the development of chronic liver disease underlying the hepatic encephalopathy phenotype." />
  <figcaption>Figure 7. Liver histopathology from bile duct-ligated mice showing fibrosis and cholestasis, confirming the development of chronic liver disease underlying the hepatic encephalopathy phenotype.<br>  Source: Wouter Claeys et al. "A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammoni." <em>Scientific reports</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36266427/">36266427</a></figcaption>
</figure>

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