Deskripsi
Immunohistochemical staining for glial fibrillary acidic protein (GFAP) in brain sections from bile duct-ligated and control mice, revealing astrocyte activation patterns associated with hepatic encephalopathy.
Figure 3
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A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammonia overload, glial cell activation and neuroinflammation.Cite This Figure
![Figure 3: Immunohistochemical staining for glial fibrillary acidic protein (GFAP) in brain sections from bile duct-ligated and control mice, revealing astrocyte activation patterns associated with hepatic encephalopathy.]() > Source: Wouter Claeys et al. "A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammoni." *Scientific reports*, 2022. PMID: [36266427](https://pubmed.ncbi.nlm.nih.gov/36266427/)
<figure> <img src="" alt="Immunohistochemical staining for glial fibrillary acidic protein (GFAP) in brain sections from bile duct-ligated and control mice, revealing astrocyte activation patterns associated with hepatic encephalopathy." /> <figcaption>Figure 3. Immunohistochemical staining for glial fibrillary acidic protein (GFAP) in brain sections from bile duct-ligated and control mice, revealing astrocyte activation patterns associated with hepatic encephalopathy.<br> Source: Wouter Claeys et al. "A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammoni." <em>Scientific reports</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36266427/">36266427</a></figcaption> </figure>