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Lipid profiling of Tecr-deficient versus wild-type brain endothelial cells, revealing altered very-long-chain fatty acid composition.

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trans-2-Enoyl-CoA Reductase Tecr-Driven Lipid Metabolism in Endothelial Cells Protects against Transcytosis to Maintain Blood-Brain Barrier Homeostasis.

Research (Washington, D.C.) (2022)

PMID: 35465346

DOI: 10.34133/2022/9839368

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![Figure 3: Lipid profiling of Tecr-deficient versus wild-type brain endothelial cells, revealing altered very-long-chain fatty acid composition.]()

> Source: Jinxuan Wang et al. "trans-2-Enoyl-CoA Reductase Tecr-Driven Lipid Metabolism in Endothelial Cells Pr." *Research (Washington, D.C.)*, 2022. PMID: [35465346](https://pubmed.ncbi.nlm.nih.gov/35465346/)

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  <img src="" alt="Lipid profiling of Tecr-deficient versus wild-type brain endothelial cells, revealing altered very-long-chain fatty acid composition." />
  <figcaption>Figure 3. Lipid profiling of Tecr-deficient versus wild-type brain endothelial cells, revealing altered very-long-chain fatty acid composition.<br>  Source: Jinxuan Wang et al. "trans-2-Enoyl-CoA Reductase Tecr-Driven Lipid Metabolism in Endothelial Cells Pr." <em>Research (Washington, D.C.)</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/35465346/">35465346</a></figcaption>
</figure>

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