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Neurotransmitter receptor antagonists (Sendide for NK1, alpha-CGRP 8-37 for CGRP, and atropine for muscarinic receptors) were tested against capsaicin-evoked thymidine kinase increases, revealing which neural pathways mediate epithelial cell renewal.

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![Figure 7: Neurotransmitter receptor antagonists (Sendide for NK1, alpha-CGRP 8-37 for CGRP, and atropine for muscarinic receptors) were tested against capsaicin-evoked thymidine kinase increases, revealing which neural pathways mediate epithelial cell renewal.]()

> Source: Ove Lundgren et al. "Intestinal epithelial stem/progenitor cells are controlled by mucosal afferent n." *PloS one*, 2011. PMID: [21347406](https://pubmed.ncbi.nlm.nih.gov/21347406/)
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  <img src="" alt="Neurotransmitter receptor antagonists (Sendide for NK1, alpha-CGRP 8-37 for CGRP, and atropine for muscarinic receptors) were tested against capsaicin-evoked thymidine kinase increases, revealing which neural pathways mediate epithelial cell renewal." />
  <figcaption>Figure 7. Neurotransmitter receptor antagonists (Sendide for NK1, alpha-CGRP 8-37 for CGRP, and atropine for muscarinic receptors) were tested against capsaicin-evoked thymidine kinase increases, revealing which neural pathways mediate epithelial cell renewal.<br>  Source: Ove Lundgren et al. "Intestinal epithelial stem/progenitor cells are controlled by mucosal afferent n." <em>PloS one</em>, 2011. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/21347406/">21347406</a></figcaption>
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