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Reactive oxygen species production rates in hippocampal synaptic mitochondria from young, middle-aged, and old rats, revealing a progressive increase in oxidative stress that precedes detectable memory impairment.
Figure 3
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Premature synaptic mitochondrial dysfunction in the hippocampus during aging contributes to memory loss.Cite This Figure
![Figure 3: Reactive oxygen species production rates in hippocampal synaptic mitochondria from young, middle-aged, and old rats, revealing a progressive increase in oxidative stress that precedes detectable memory impairment.]() > Source: Margrethe A Olesen et al. "Premature synaptic mitochondrial dysfunction in the hippocampus during aging con." *Redox biology*, 2020. PMID: [32447261](https://pubmed.ncbi.nlm.nih.gov/32447261/)
<figure> <img src="" alt="Reactive oxygen species production rates in hippocampal synaptic mitochondria from young, middle-aged, and old rats, revealing a progressive increase in oxidative stress that precedes detectable memory impairment." /> <figcaption>Figure 3. Reactive oxygen species production rates in hippocampal synaptic mitochondria from young, middle-aged, and old rats, revealing a progressive increase in oxidative stress that precedes detectable memory impairment.<br> Source: Margrethe A Olesen et al. "Premature synaptic mitochondrial dysfunction in the hippocampus during aging con." <em>Redox biology</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32447261/">32447261</a></figcaption> </figure>