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Integrated model depicting how premature synaptic mitochondrial dysfunction in the hippocampus drives age-related memory loss, connecting bioenergetic failure, calcium dysregulation, oxidative stress, and synaptic transmission impairment.

Figure 11

Diagram

Source Paper

Premature synaptic mitochondrial dysfunction in the hippocampus during aging contributes to memory loss.

Redox biology (2020)

PMID: 32447261

DOI: 10.1016/j.redox.2020.101558

Cite This Figure

![Figure 11: Integrated model depicting how premature synaptic mitochondrial dysfunction in the hippocampus drives age-related memory loss, connecting bioenergetic failure, calcium dysregulation, oxidative stress, and synaptic transmission impairment.]()

> Source: Margrethe A Olesen et al. "Premature synaptic mitochondrial dysfunction in the hippocampus during aging con." *Redox biology*, 2020. PMID: [32447261](https://pubmed.ncbi.nlm.nih.gov/32447261/)

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  <img src="" alt="Integrated model depicting how premature synaptic mitochondrial dysfunction in the hippocampus drives age-related memory loss, connecting bioenergetic failure, calcium dysregulation, oxidative stress, and synaptic transmission impairment." />
  <figcaption>Figure 11. Integrated model depicting how premature synaptic mitochondrial dysfunction in the hippocampus drives age-related memory loss, connecting bioenergetic failure, calcium dysregulation, oxidative stress, and synaptic transmission impairment.<br>  Source: Margrethe A Olesen et al. "Premature synaptic mitochondrial dysfunction in the hippocampus during aging con." <em>Redox biology</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32447261/">32447261</a></figcaption>
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