Mô tả

Proposed molecular pathway through which BDMC antagonizes Alzheimer's disease involves SIRT1 upregulation, reduced oxidative stress, and inhibition of amyloid-beta-induced apoptosis. The SIRT1/Nrf2 axis is a key mediator.

Figure 6

Diagram

Source Paper

Bisdemethoxycurcumin inhibits oxidative stress and antagonizes Alzheimer's disease by up-regulating SIRT1.

Brain and behavior (2020)

PMID: 32441492

DOI: 10.1002/brb3.1655

Cite This Figure

![Figure 6: Proposed molecular pathway through which BDMC antagonizes Alzheimer's disease involves SIRT1 upregulation, reduced oxidative stress, and inhibition of amyloid-beta-induced apoptosis. The SIRT1/Nrf2 axis is a key mediator.]()

> Source: Yan Xu et al. "Bisdemethoxycurcumin inhibits oxidative stress and antagonizes Alzheimer's disea." *Brain and behavior*, 2020. PMID: [32441492](https://pubmed.ncbi.nlm.nih.gov/32441492/)

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  <img src="" alt="Proposed molecular pathway through which BDMC antagonizes Alzheimer's disease involves SIRT1 upregulation, reduced oxidative stress, and inhibition of amyloid-beta-induced apoptosis. The SIRT1/Nrf2 axis is a key mediator." />
  <figcaption>Figure 6. Proposed molecular pathway through which BDMC antagonizes Alzheimer's disease involves SIRT1 upregulation, reduced oxidative stress, and inhibition of amyloid-beta-induced apoptosis. The SIRT1/Nrf2 axis is a key mediator.<br>  Source: Yan Xu et al. "Bisdemethoxycurcumin inhibits oxidative stress and antagonizes Alzheimer's disea." <em>Brain and behavior</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32441492/">32441492</a></figcaption>
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