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Oxidative stress markers in vascular endothelium of MTHFR-deficient models, comparing untreated and SIRT1-activator-treated conditions. The findings indicate that SIRT1 activation may attenuate oxidative damage associated with MTHFR polymorphism.
Figure 7
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SIRT1 pharmacological activation rescues vascular dysfunction and prevents thrombosis in MTHFR deficiency.Cite This Figure
![Figure 7: Oxidative stress markers in vascular endothelium of MTHFR-deficient models, comparing untreated and SIRT1-activator-treated conditions. The findings indicate that SIRT1 activation may attenuate oxidative damage associated with MTHFR polymorphism.]() > Source: Albino Carrizzo et al. "SIRT1 pharmacological activation rescues vascular dysfunction and prevents throm." *Cellular and molecular life sciences : CMLS*, 2022. PMID: [35821533](https://pubmed.ncbi.nlm.nih.gov/35821533/)
<figure> <img src="" alt="Oxidative stress markers in vascular endothelium of MTHFR-deficient models, comparing untreated and SIRT1-activator-treated conditions. The findings indicate that SIRT1 activation may attenuate oxidative damage associated with MTHFR polymorphism." /> <figcaption>Figure 7. Oxidative stress markers in vascular endothelium of MTHFR-deficient models, comparing untreated and SIRT1-activator-treated conditions. The findings indicate that SIRT1 activation may attenuate oxidative damage associated with MTHFR polymorphism.<br> Source: Albino Carrizzo et al. "SIRT1 pharmacological activation rescues vascular dysfunction and prevents throm." <em>Cellular and molecular life sciences : CMLS</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/35821533/">35821533</a></figcaption> </figure>