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HMGB1 (high-mobility group box 1) release from neurons exposed to MPO-derived HOCl, suggesting a mechanism for amplifying brain damage during ischemia-reperfusion.
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Hypochlorous acid derived from microglial myeloperoxidase could mediate high-mobility group box 1 release from neurons to amplify brain damage in …Cite This Figure
![Figure 4: HMGB1 (high-mobility group box 1) release from neurons exposed to MPO-derived HOCl, suggesting a mechanism for amplifying brain damage during ischemia-reperfusion.]() > Source: Shuang Chen et al. "Hypochlorous acid derived from microglial myeloperoxidase could mediate high-mob." *Journal of neuroinflammation*, 2024. PMID: [38515139](https://pubmed.ncbi.nlm.nih.gov/38515139/)
<figure> <img src="" alt="HMGB1 (high-mobility group box 1) release from neurons exposed to MPO-derived HOCl, suggesting a mechanism for amplifying brain damage during ischemia-reperfusion." /> <figcaption>Figure 4. HMGB1 (high-mobility group box 1) release from neurons exposed to MPO-derived HOCl, suggesting a mechanism for amplifying brain damage during ischemia-reperfusion.<br> Source: Shuang Chen et al. "Hypochlorous acid derived from microglial myeloperoxidase could mediate high-mob." <em>Journal of neuroinflammation</em>, 2024. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/38515139/">38515139</a></figcaption> </figure>