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Lipid peroxidation levels (4-HNE and MDA) in synaptic mitochondrial fractions from hippocampus, demonstrating elevated oxidative damage to membrane lipids in aged animals compared to young controls.
Figure 9
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Premature synaptic mitochondrial dysfunction in the hippocampus during aging contributes to memory loss.Cite This Figure
![Figure 9: Lipid peroxidation levels (4-HNE and MDA) in synaptic mitochondrial fractions from hippocampus, demonstrating elevated oxidative damage to membrane lipids in aged animals compared to young controls.]() > Source: Margrethe A Olesen et al. "Premature synaptic mitochondrial dysfunction in the hippocampus during aging con." *Redox biology*, 2020. PMID: [32447261](https://pubmed.ncbi.nlm.nih.gov/32447261/)
<figure> <img src="" alt="Lipid peroxidation levels (4-HNE and MDA) in synaptic mitochondrial fractions from hippocampus, demonstrating elevated oxidative damage to membrane lipids in aged animals compared to young controls." /> <figcaption>Figure 9. Lipid peroxidation levels (4-HNE and MDA) in synaptic mitochondrial fractions from hippocampus, demonstrating elevated oxidative damage to membrane lipids in aged animals compared to young controls.<br> Source: Margrethe A Olesen et al. "Premature synaptic mitochondrial dysfunction in the hippocampus during aging con." <em>Redox biology</em>, 2020. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/32447261/">32447261</a></figcaption> </figure>