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Iron chelation capacity of ALA is quantified in vitro, showing effective reduction of intracellular iron accumulation in ferric ammonium citrate (FAC)-treated microglia.

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Source Paper

Neuroprotective Role of α-Lipoic Acid in Iron-Overload-Mediated Toxicity and Inflammation in In Vitro and In Vivo Models.

Antioxidants (Basel, Switzerland) (2022)

PMID: 36009316

DOI: 10.3390/antiox11081596

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![Figure 2: Iron chelation capacity of ALA is quantified in vitro, showing effective reduction of intracellular iron accumulation in ferric ammonium citrate (FAC)-treated microglia.]()

> Source: Giuseppe Carota et al. "Neuroprotective Role of α-Lipoic Acid in Iron-Overload-Mediated Toxicity and Inf." *Antioxidants (Basel, Switzerland)*, 2022. PMID: [36009316](https://pubmed.ncbi.nlm.nih.gov/36009316/)

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  <img src="" alt="Iron chelation capacity of ALA is quantified in vitro, showing effective reduction of intracellular iron accumulation in ferric ammonium citrate (FAC)-treated microglia." />
  <figcaption>Figure 2. Iron chelation capacity of ALA is quantified in vitro, showing effective reduction of intracellular iron accumulation in ferric ammonium citrate (FAC)-treated microglia.<br>  Source: Giuseppe Carota et al. "Neuroprotective Role of α-Lipoic Acid in Iron-Overload-Mediated Toxicity and Inf." <em>Antioxidants (Basel, Switzerland)</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36009316/">36009316</a></figcaption>
</figure>

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