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368 figures issues de recherches évaluées par des pairs

Tous Vitamin E Green Tea Extract (EGCG) Citicoline Folate Zinc Bacopa monnieri Omega-3 Fatty Acids (DHA/EPA) Alpha-Lipoic Acid Creatine Resveratrol Vitamin D L-Theanine Vitamin B12 Ginkgo biloba Lutein & Zeaxanthin Melatonin Rhodiola rosea Panax Ginseng Phosphatidylserine Taurine Curcumin Uridine Monophosphate
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Figure 1. Flow-chart.
Figure 1

Figure 1. Flow-chart.

Exploring the Efficacy and Safety of Nutritional Supplements in Alzheimer's Disease.

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Figure 2

Exploring the Efficacy and Safety of Nutritional Supplements in Alzheimer's Disease.

Figure 13
Figure 13

Mitigating Age-Related Cognitive Decline and Oxidative Status in Rats Treated with Catechin …

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Figure 14

Mitigating Age-Related Cognitive Decline and Oxidative Status in Rats Treated with Catechin …

Figure 1
Figure 1

The Role of Omega-3 Fatty Acid Supplementation in Slowing Cognitive Decline Among …

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Figure 4

Concentration of Phosphatidylserine Influence Rates of Insulin Aggregation and Toxicity of Amyloid …

Figure 1. Increase in the concentration of PS in the lipid mixtures increases the aggregation rate of insulin. Averages of triplicates of ThT aggregation kinetics of insulin (Ins) in the lipid-free environment (red), insulin in the presence of LUVs of PC/
Figure 6

Figure 1. Increase in the concentration of PS in the lipid mixtures increases the aggregation rate of insulin. Averages of triplicates of ThT aggregation kinetics of insulin (Ins) in the …

Concentration of Phosphatidylserine Influence Rates of Insulin Aggregation and Toxicity of Amyloid …

Figure 7
Figure 7

Concentration of Phosphatidylserine Influence Rates of Insulin Aggregation and Toxicity of Amyloid …

Figure 4. AFM-IR spectra acquired from insulin (Ins) fibrils grown in the lipid-free environment (red), insulin in the presence of LUVs of PC/PE/PS (40:40:20) (yellow), PC/PE/PS (30:40:30) (green), PC/ PE/PS (20:40:40) (blue), and PC/PE/PS (30:30:40) (pur
Figure 8

Figure 4. AFM-IR spectra acquired from insulin (Ins) fibrils grown in the lipid-free environment (red), insulin in the presence of LUVs of PC/PE/PS (40:40:20) (yellow), PC/PE/PS (30:40:30) (green), PC/ PE/PS …

Concentration of Phosphatidylserine Influence Rates of Insulin Aggregation and Toxicity of Amyloid …

Figure 9
Figure 9

Concentration of Phosphatidylserine Influence Rates of Insulin Aggregation and Toxicity of Amyloid …

Figure 10
Figure 10

Concentration of Phosphatidylserine Influence Rates of Insulin Aggregation and Toxicity of Amyloid …

Figure 1. Flow diagram of PubMed search for choline in parenteral nutrition. Please refer to the text for references.
Figure 2

Figure 1. Flow diagram of PubMed search for choline in parenteral nutrition. Please refer to the text for references.

Evidence and Perspectives for Choline Supplementation during Parenteral Nutrition-A Narrative Review.

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Figure 3

Evidence and Perspectives for Choline Supplementation during Parenteral Nutrition-A Narrative Review.

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Figure 4

Evidence and Perspectives for Choline Supplementation during Parenteral Nutrition-A Narrative Review.

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Figure 5

Evidence and Perspectives for Choline Supplementation during Parenteral Nutrition-A Narrative Review.

Figure 1. Iron metabolism in the enterocyte and hemochromatosis. (A) Iron is absorbed as Fe2+ through reduction by DcytB/STEAP, and transported via DMT1. In the case iron is bound to heme, it can be transported via heme carrier 1. Another route is via rec
Figure 2

Figure 1. Iron metabolism in the enterocyte and hemochromatosis. (A) Iron is absorbed as Fe2+ through reduction by DcytB/STEAP, and transported via DMT1. In the case iron is bound to …

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 2. Zinc metabolism in the enterocyte and its overload. (A) Zinc is absorbed as Zn2+ via the ZIPs (mainly ZIP4) and can be excreted into the intestinal lumen by the ZnTs. In the enterocytes, Zn2+ can be found as cytoplasmic free zinc (which can bind
Figure 3

Figure 2. Zinc metabolism in the enterocyte and its overload. (A) Zinc is absorbed as Zn2+ via the ZIPs (mainly ZIP4) and can be excreted into the intestinal lumen by …

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 4
Figure 4

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 3. Copper metabolism in the enterocytes and Wilson disease. (A) Copper is reduced to Cu+ by STEAP or DcytB, and transported mainly via CTR1, but also by DMT1/CTR2. Thereupon,
Figure 5

Figure 3. Copper metabolism in the enterocytes and Wilson disease. (A) Copper is reduced to Cu+ by STEAP or DcytB, and transported mainly via CTR1, but also by DMT1/CTR2. Thereupon,

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 6
Figure 6

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 4. Manganese metabolism and hypermanganesemia. (A) Manganese can be taken up as Mn2+ via DMT1, ZIP, or Ca2+ channels, or as Mn3+ by binding to transferrin. In mitochondria, Mn2+ works as a cofactor for MnSOD (SOD2). Manganese (Mn2+) can also influe
Figure 7

Figure 4. Manganese metabolism and hypermanganesemia. (A) Manganese can be taken up as Mn2+ via DMT1, ZIP, or Ca2+ channels, or as Mn3+ by binding to transferrin. In mitochondria, Mn2+ …

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 5. Overview of cellular mechanisms related to toxicity by essential transition metals. (A) Different metabolic pathways in the cell use O2, e.g., oxidative phosphorylation in the mitochondria. The generation of ROS, such as H2O2 and •O−
Figure 8

Figure 5. Overview of cellular mechanisms related to toxicity by essential transition metals. (A) Different metabolic pathways in the cell use O2, e.g., oxidative phosphorylation in the mitochondria. The generation …

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 9
Figure 9

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

Figure 10
Figure 10

Metabolic Derangement of Essential Transition Metals and Potential Antioxidant Therapies.

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