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BrainCited

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5 figure da ricerca revisionata da esperti

Tutti Omega-3 Fatty Acids (DHA/EPA) Bacopa monnieri Citicoline Phosphatidylserine Ginkgo biloba Curcumin Creatine Rhodiola rosea L-Theanine Panax Ginseng Vitamin D Vitamin B12 Folate Resveratrol Alpha-Lipoic Acid Vitamin E Green Tea Extract (EGCG) Zinc Uridine Monophosphate Taurine Lutein & Zeaxanthin Melatonin
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Figure 1. Western blot analyses of protein markers in fractions 1–6 from NSL, PD and PDi brain cortices. Equal amounts of total protein were used for NSL, PD and iPD samples. Standard molecular weight values are indicated (left).
Figure 1 Photograph

Western blot analyses of protein markers across density gradient fractions from control, Parkinson's disease, and incidental PD brain cortices reveal altered lipid raft protein distribution.

Severe alterations in lipid composition of frontal cortex lipid rafts from Parkinson's …

Figure 3
Figure 3 Photograph

Brain imaging studies demonstrate that elevated homocysteine is associated with accelerated rates of brain atrophy, particularly in medial temporal lobe structures critical for memory. B-vitamin treatment appears to slow this atrophy in individuals with elevated baseline homocysteine.

Homocysteine and Dementia: An International Consensus Statement.

Figure 13
Figure 13 Photograph

Brain imaging in neuroacanthocytosis typically reveals caudate nucleus atrophy and putaminal changes. This figure presents structural neuroimaging findings characteristic of advanced basal ganglia degeneration in NA patients.

Neuroacanthocytosis syndromes.

Figure 14
Figure 14 Photograph

Brain imaging in neuroacanthocytosis typically reveals caudate nucleus atrophy and putaminal changes. This figure presents structural neuroimaging findings characteristic of advanced basal ganglia degeneration in NA patients.

Neuroacanthocytosis syndromes.

Figure 15
Figure 15 Photograph

Brain imaging in neuroacanthocytosis typically reveals caudate nucleus atrophy and putaminal changes. This figure presents structural neuroimaging findings characteristic of advanced basal ganglia degeneration in NA patients.

Neuroacanthocytosis syndromes.