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Araştırma Süreci

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Tümü Vitamin E Green Tea Extract (EGCG) Citicoline Folate Zinc Bacopa monnieri Omega-3 Fatty Acids (DHA/EPA) Alpha-Lipoic Acid Creatine Resveratrol Vitamin D L-Theanine Vitamin B12 Ginkgo biloba Lutein & Zeaxanthin Melatonin Rhodiola rosea Panax Ginseng Phosphatidylserine Taurine Curcumin Uridine Monophosphate
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Figure 1. Western blot analyses of protein markers in fractions 1–6 from NSL, PD and PDi brain cortices. Equal amounts of total protein were used for NSL, PD and iPD samples. Standard molecular weight values are indicated (left).
Figure 1 Photograph

Western blot analyses of protein markers across density gradient fractions from control, Parkinson's disease, and incidental PD brain cortices reveal altered lipid raft protein distribution.

Severe alterations in lipid composition of frontal cortex lipid rafts from Parkinson's …

Figure 3
Figure 3 Photograph

Brain imaging studies demonstrate that elevated homocysteine is associated with accelerated rates of brain atrophy, particularly in medial temporal lobe structures critical for memory. B-vitamin treatment appears to slow this atrophy in individuals with elevated baseline homocysteine.

Homocysteine and Dementia: An International Consensus Statement.

Figure 13
Figure 13 Photograph

Brain imaging in neuroacanthocytosis typically reveals caudate nucleus atrophy and putaminal changes. This figure presents structural neuroimaging findings characteristic of advanced basal ganglia degeneration in NA patients.

Neuroacanthocytosis syndromes.

Figure 14
Figure 14 Photograph

Brain imaging in neuroacanthocytosis typically reveals caudate nucleus atrophy and putaminal changes. This figure presents structural neuroimaging findings characteristic of advanced basal ganglia degeneration in NA patients.

Neuroacanthocytosis syndromes.

Figure 15
Figure 15 Photograph

Brain imaging in neuroacanthocytosis typically reveals caudate nucleus atrophy and putaminal changes. This figure presents structural neuroimaging findings characteristic of advanced basal ganglia degeneration in NA patients.

Neuroacanthocytosis syndromes.