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BrainCited

연구 프로세스

368 동료 심사 연구의 그림

전체 Vitamin E Green Tea Extract (EGCG) Citicoline Folate Zinc Bacopa monnieri Omega-3 Fatty Acids (DHA/EPA) Alpha-Lipoic Acid Creatine Resveratrol Vitamin D L-Theanine Vitamin B12 Ginkgo biloba Lutein & Zeaxanthin Melatonin Rhodiola rosea Panax Ginseng Phosphatidylserine Taurine Curcumin Uridine Monophosphate
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Figure 1. Effects of C. butyricum and 25-hydroxyvitamin D3 on the latency-to-lie time, tibial content of calcium and phosphorus, BMD and bone-breaking strengthen of broilers. (A) LTL. (B) calcium. (C) phosphorus. (D) BMD. (E) bone-breaking strengthen. (F)
Figure 1 Chart

Effects of dietary Clostridium butyricum and 25-hydroxyvitamin D3 supplementation on latency-to-lie time in a poultry model, indicating improvements in leg health and bone strength.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 2
Figure 2 Chart

Experimental data from a study on dietary Clostridium butyricum and 25-hydroxyvitamin D3 and their combined effects on bone metabolism through gut microbiota modulation in poultry.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 4. Effects of Clostridium butyricum and 25-hydroxyvitamin D3 on the hypothalamic and intestinal brain-gut peptides in broilers. (A) Caecal 5-HT content. (B) Caecal DA content. (C) Caecal GLP-1 content. (D) Ileal PYY content. (E) Hypothalamic 5-HT c
Figure 3 Chart

Hypothalamic signaling pathway analysis in poultry fed Clostridium butyricum and 25-hydroxyvitamin D3, examining effects on bone metabolism regulatory mechanisms.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 5. Effects of Clostridium butyricum and 25-hydroxyvitamin D3 on caecal SCFAs in broilers. (A) acetic acid level. (B) propionic acid level. (C) isobutyric level. (D) butyric level. (E) isovaleric level. (F) valeric level. Con birds fed basal diet wi
Figure 4 Chart

Caecal short-chain fatty acid (SCFA) concentrations in poultry supplemented with Clostridium butyricum and 25-hydroxyvitamin D3, linking gut fermentation products to bone metabolism modulation.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 6. Effects of Clostridium butyricum and 25-hydroxyvitamin D3 on metagenome of broilers’ caecal microflora. (A) gene number venn graph. (B) core_Pan gene dilution curve. (C) species relative abundance histogram display based on genus level. (D) speci
Figure 5 Chart

Metagenomic analysis of caecal microbiota in poultry receiving Clostridium butyricum and 25-hydroxyvitamin D3 supplementation, revealing shifts in microbial community structure.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 7. Effects of Clostridium butyricum and 25-hydroxyvitamin D3 on caecal microflora on genus and species levels in broilers. (A) top 12 distinguished genus based on Kruskal_Wallis analysis; (B) top 12 distinguished species based on Kruskal_Wallis anal
Figure 6 Chart

Caecal microbial composition data from poultry treated with Clostridium butyricum and 25-hydroxyvitamin D3, showing taxonomic-level changes associated with improved bone metabolism.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 8. Effects of Clostridium butyricum and 25-hydroxyvitamin D3 on KEGG metabolic pathways of caecal microflora in broilers based on metagenomics. (A) Functional level PCA analysis. (B) Functional level PCoA analysis. (C) Heatmap of KEGG pathways. (D)
Figure 7 Chart

Metagenomic analysis of caecal microbiota in poultry receiving Clostridium butyricum and 25-hydroxyvitamin D3 supplementation, revealing shifts in microbial community structure.

Dietary Clostridium butyricum and 25-Hydroxyvitamin D3 modulate bone metabolism of broilers through …

Figure 4
Figure 4 Chart

Electrophysiology or binding affinity data for propofol at different GABAA receptor binding sites, comparing potency across the three identified site classes.

Three classes of propofol binding sites on GABAA receptors.

Figure 5
Figure 5 Chart

Mutagenesis or competition binding data supporting the classification of three distinct propofol binding site classes on GABAA receptors.

Three classes of propofol binding sites on GABAA receptors.

Figure 6
Figure 6 Chart

Structure-activity relationship analysis of propofol analogs at the different GABAA receptor binding sites, informing anesthetic drug design.

Three classes of propofol binding sites on GABAA receptors.

Figure 7
Figure 7 Chart

Molecular dynamics simulation or computational modeling of propofol interactions with GABAA receptor transmembrane domains.

Three classes of propofol binding sites on GABAA receptors.

Figure 8
Figure 8 Chart

Comparative analysis of propofol binding site occupancy and functional consequences for GABAA receptor channel gating and anesthetic efficacy.

Three classes of propofol binding sites on GABAA receptors.

Figure 9
Figure 9 Chart

Supplementary structural or pharmacological data supporting the three-class model of propofol binding to GABAA receptors.

Three classes of propofol binding sites on GABAA receptors.

Figure 7. Synthetic scheme for synthesis of cumene-d11 from benzene-d6.
Figure 10 Diagram

Synthetic scheme for the preparation of cumene-d11 from benzene-d6, used as a deuterated propofol analog for binding site characterization studies on GABAA receptors.

Three classes of propofol binding sites on GABAA receptors.

Figure 1. Flow chart diagram illustrating the database searches, number of publications identified, screened, and final full texts included in the systematic review.
Figure 2

Figure 1. Flow chart diagram illustrating the database searches, number of publications identified, screened, and final full texts included in the systematic review.

The Role of Minoxidil in Treatment of Alopecia Areata: A Systematic Review …

Figure 3
Figure 3

The Role of Minoxidil in Treatment of Alopecia Areata: A Systematic Review …

In total, 164 patients treated with less than 5% minoxidil were included in the metaanalysis. For the group of patients using minoxidil at a concentration lower than 5%, the response rate was 58% (95% Cl 0.5–0.67), and the prediction interval for the vari
Figure 4

In total, 164 patients treated with less than 5% minoxidil were included in the metaanalysis. For the group of patients using minoxidil at a concentration lower than 5%, the response …

The Role of Minoxidil in Treatment of Alopecia Areata: A Systematic Review …

Figure 5
Figure 5

The Role of Minoxidil in Treatment of Alopecia Areata: A Systematic Review …

Figure 1
Figure 1 Chart

Behavioral or neurological assessment data from a study evaluating alpha-lipoic acid's neuroprotective effects against dapsone-induced neuroinflammation and oxidative stress in an animal model.

Alpha-Lipoic Acid Reduces Neuroinflammation and Oxidative Stress Induced by Dapsone in an …

Figure 2
Figure 2 Chart

Brain tissue analysis showing markers of neuroinflammation in dapsone-treated animals, comparing alpha-lipoic acid-supplemented versus control groups.

Alpha-Lipoic Acid Reduces Neuroinflammation and Oxidative Stress Induced by Dapsone in an …

Figure 3. Effects of dapsone (40 mg/kg; ip) on the levels of cytokines IL-1β, IL-17 and IL-4 and BDNF in the prefrontal cortex (PFC) of mice and post-treatment with ALA (25 mg/kg) for 5 consecutive days. (A) Concentration of IL1-β (pg/mL), (B) concentrati
Figure 3 Chart

Cytokine levels (IL-1beta, IL-6, and related markers) in prefrontal cortex tissue of dapsone-treated animals, demonstrating alpha-lipoic acid's anti-inflammatory effects.

Alpha-Lipoic Acid Reduces Neuroinflammation and Oxidative Stress Induced by Dapsone in an …

Figure 4
Figure 4 Micrograph

Histological or immunohistochemical analysis of brain tissue from the alpha-lipoic acid and dapsone neuroinflammation study.

Alpha-Lipoic Acid Reduces Neuroinflammation and Oxidative Stress Induced by Dapsone in an …

Figure 5. Effects of dapsone (40 mg/kg; ip) on TEAC, GSH, SOD and CAT in the prefrontal cortex and hippocampus of mice and post-treatment with ALA (25 mg/kg) for 5 consecutive days. (A) Concentration of TEAC (mmol/L), (B) concentration of GSH (μmol/mL), (
Figure 5 Chart

Antioxidant capacity measurements (TEAC, GSH, SOD, and CAT) in prefrontal cortex tissue, showing alpha-lipoic acid's ability to counteract dapsone-induced oxidative stress.

Alpha-Lipoic Acid Reduces Neuroinflammation and Oxidative Stress Induced by Dapsone in an …

Figure 6. Effects of dapsone (40 mg/kg) on the generation of thiobarbituric acid reactive substances (TBARS) and iron concentration in the prefrontal cortex (PFC) and hippocampus of mice and posttreatment with ALA (25 mg/kg) for 5 consecutive days. (A) Co
Figure 6 Chart

Thiobarbituric acid reactive substances (TBARS) measurements indicating lipid peroxidation levels in dapsone-treated animals, with and without alpha-lipoic acid intervention.

Alpha-Lipoic Acid Reduces Neuroinflammation and Oxidative Stress Induced by Dapsone in an …

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