Mô tả

Iron metabolism gene expression (HO-1, FPN1, DMT1) in HMC3 cells is shown after FAC and ALA treatment. ALA prevents the upregulation of iron uptake and metabolism mediators induced by iron overload.

Figure 6

Chart

Source Paper

Neuroprotective Role of α-Lipoic Acid in Iron-Overload-Mediated Toxicity and Inflammation in In Vitro and In Vivo Models.

Antioxidants (Basel, Switzerland) (2022)

PMID: 36009316

DOI: 10.3390/antiox11081596

Cite This Figure

![Figure 6: Iron metabolism gene expression (HO-1, FPN1, DMT1) in HMC3 cells is shown after FAC and ALA treatment. ALA prevents the upregulation of iron uptake and metabolism mediators induced by iron overload.]()

> Source: Giuseppe Carota et al. "Neuroprotective Role of α-Lipoic Acid in Iron-Overload-Mediated Toxicity and Inf." *Antioxidants (Basel, Switzerland)*, 2022. PMID: [36009316](https://pubmed.ncbi.nlm.nih.gov/36009316/)

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  <img src="" alt="Iron metabolism gene expression (HO-1, FPN1, DMT1) in HMC3 cells is shown after FAC and ALA treatment. ALA prevents the upregulation of iron uptake and metabolism mediators induced by iron overload." />
  <figcaption>Figure 6. Iron metabolism gene expression (HO-1, FPN1, DMT1) in HMC3 cells is shown after FAC and ALA treatment. ALA prevents the upregulation of iron uptake and metabolism mediators induced by iron overload.<br>  Source: Giuseppe Carota et al. "Neuroprotective Role of α-Lipoic Acid in Iron-Overload-Mediated Toxicity and Inf." <em>Antioxidants (Basel, Switzerland)</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36009316/">36009316</a></figcaption>
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