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BrainCited

Resveratrol 图表

11 来自同行评审研究的图表

全部 Vitamin E Green Tea Extract (EGCG) Citicoline Folate Zinc Bacopa monnieri Omega-3 Fatty Acids (DHA/EPA) Alpha-Lipoic Acid Creatine Resveratrol Vitamin D L-Theanine Vitamin B12 Ginkgo biloba Lutein & Zeaxanthin Melatonin Rhodiola rosea Panax Ginseng Phosphatidylserine Taurine Curcumin Uridine Monophosphate
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Figure 5
Figure 5 Chart

Vascular biomarkers and endothelial function parameters were measured to assess resveratrol's cardiovascular effects. This figure presents changes in vascular health indicators during the 14-week supplementation trial.

Effects of Resveratrol on Cognitive Performance, Mood and Cerebrovascular Function in Post-Menopausal …

Figure 2. Effects of resveratrol on individual cognitive tests and overcall cognitive performance. Pre-post intervention differences in cognitive performance were calculated for each group using Z-scores. Significant differences between resveratrol and pla
Figure 6 Chart

Resveratrol supplementation was associated with changes in cognitive test performance in post-menopausal women. Pre-post intervention differences calculated using Z-scores revealed significant differences between resveratrol and placebo groups on individual cognitive domains and overall cognitive performance.

Effects of Resveratrol on Cognitive Performance, Mood and Cerebrovascular Function in Post-Menopausal …

Figure 3. Cerebrovascular responsiveness to hypercapnia in the middle cerebral artery (MCA). Transcranial Doppler (TCD) ultrasound was used to measure blood flow velocity in the MCA in order to calculate treatment changes after 14 weeks in CVR to hypercapn
Figure 7 Chart

Cerebrovascular responsiveness to hypercapnia was measured in the middle cerebral artery using transcranial Doppler ultrasound. Treatment changes in cerebrovascular reactivity after 14 weeks suggest resveratrol may enhance cerebral blood flow regulation in post-menopausal women.

Effects of Resveratrol on Cognitive Performance, Mood and Cerebrovascular Function in Post-Menopausal …

Figure 2
Figure 2 Chart

Bone mineral density measurements at key skeletal sites are compared between resveratrol and placebo groups over the trial period. Resveratrol, a polyphenol found in red grapes, was hypothesized to act as a phytoestrogen with bone-protective properties.

Regular Supplementation With Resveratrol Improves Bone Mineral Density in Postmenopausal Women: A …

Figure 3
Figure 3 Chart

Secondary outcomes or biomarker data from the resveratrol supplementation trial are displayed. The analysis examines whether improvements in bone mineral density correlate with changes in bone turnover markers or inflammatory mediators.

Regular Supplementation With Resveratrol Improves Bone Mineral Density in Postmenopausal Women: A …

Figure 5
Figure 5 Chart

Subcellular distribution of PKC isoforms between cytosolic and membrane fractions is analyzed, showing altered translocation patterns in aged brain tissue.

Age-Dependent Levels of Protein Kinase Cs in Brain: Reduction of Endogenous Mechanisms …

Figure 6
Figure 6 Chart

RACK1 and RACK2 scaffolding protein levels are measured in aging hippocampus, indicating reduced anchoring capacity for activated PKC isoforms.

Age-Dependent Levels of Protein Kinase Cs in Brain: Reduction of Endogenous Mechanisms …

Figure 2. γPKC, δPKC and εPKC levels in hippocampus of young (Y), middle-aged (M), and aged rats (O). Western Blot analysis of γPKC, δPKC and εPKC levels in the soluble and particulate fractions of young, middle-aged, and old rats hippocampus. γPKC levels
Figure 7 Chart

Western blot analysis of gamma, delta, and epsilon PKC levels in hippocampus across young, middle-aged, and aged rats demonstrates progressive age-related reduction.

Age-Dependent Levels of Protein Kinase Cs in Brain: Reduction of Endogenous Mechanisms …

Figure 8
Figure 8 Chart

Quantitative densitometry of PKC isoform bands confirms statistically significant declines in neuroprotective PKC levels with advancing age.

Age-Dependent Levels of Protein Kinase Cs in Brain: Reduction of Endogenous Mechanisms …

Since activation of PKCs leads to their translocation to the particulate fraction, where they bind with their respective RACKs, we next measured RACK1 and RACK2 levels. RACK1 (58.53 ± 7.2, p < 0.05) and RACK2 (34.14 ± 8.2, p < 0.02) levels in the ag
Figure 9 Chart

Activated PKC translocation to membrane fractions is diminished in aged rats, with reduced binding to RACK scaffolding proteins contributing to impaired neuroprotective signaling.

Age-Dependent Levels of Protein Kinase Cs in Brain: Reduction of Endogenous Mechanisms …

Figure 3. RACK1 and RACK2 levels in the hippocampus of young, middle-aged, and aged rats. Western Blot analysis of RACK1 (a) and RACK2 (b) expression in young (Y), middle aged (M) and old (O) rats hippocampus. RACK1 and RACK2 levels differed significantly b
Figure 11 Chart

RACK1 and RACK2 protein levels in hippocampal tissue are quantified by Western blot across three age groups, with both scaffolding proteins showing age-related reductions.

Age-Dependent Levels of Protein Kinase Cs in Brain: Reduction of Endogenous Mechanisms …